An anomalous origin of the left coronary artery arising from the pulmonary artery (ALCAPA) is an uncommon congenital coronary abnormality, occurring in fewer than 0.01% of patients undergoing diagnostic cardiac catheterization.1,2 Also termed Bland-White-Garland syndrome,2 ALCAPA is characterized by blood flow passing from a dilated right coronary artery to the left coronary arteries by collateral vessels and then passing retrograde to exit the anomalous left main artery into the pulmonary artery.
Usually identified during infancy or early childhood, ALCAPA is clinically manifest as ischemic

In this month's issue of the Journal of Invasive Cardiology, a new section dedicated to the invasive and interventional management of acute coronary syndromes (ACS) is being launched. This is an exciting opportunity for our community to share the latest observations in pathogenesis, treatment strategies and leading edge pharmacologic adjuncts useful in the therapy of patients with acute chest pain.
It is a dream role for me to serve as Section Editor because of my dual interests in interventional cardiology and management of acute ischemia. My goal is to develop a different kind of education

The acute coronary syndromes share a common pathogenetic mechanism involving plaque disruption, platelet activation and superimposed thrombus.1 Acutely ruptured plaques have a characteristic angiographic appearance directly reflecting mechanical disruption at the shoulder of the atherosclerotic plaque. Intracoronary thrombus is also commonly observed in patients with severe episodes of acute ischemia and is angiographically demonstrable with a higher incidence than in less acute presentations.2 The marked heterogeneity in patient characteristics and morphologic features directly reflects the w